I have Native Access installed along with a cracked copy of Kontakt 5 and also have a serial number for Kontakt 5 Library. I have been searching for days for a solution to this problem and have tried every combination of things I have seen on the internet. I have even tried another serial number from another version of Kontakt 5 and it still gives me this error. I just want to use this library in my Kontakt 5 Library and was hoping to be able to do that.
A:
I used the answer from 'TheWellSmurf' and had the same error and fixed it. I had to reset my serial number and use a different one. So the issue is that you need a different serial number if you use the Library with Native Access.
Aortic hemodynamics in rats exposed to chronic intermittent hypoxia for 4 months.
The rat model has been used extensively for the study of pathophysiology and for the development of therapeutic approaches for human diseases. Although previous studies have demonstrated that chronic intermittent hypoxia (CIH) in rats increases blood pressure (BP) and vascular function, there is no clear evidence of the effects of CIH on aortic hemodynamics. In this study, we measured aortic hemodynamic parameters and ventilatory response to hypoxia in CIH rats. Eight-week-old rats were exposed to CIH for 4 months (2 h hypoxia, 10% O(2), 10% CO(2) in N(2)) and a control group was kept in normoxia. Aortic BP, heart rate (HR), left ventricular end-diastolic pressure (LVEDP), and cardiac output (CO) were measured by high-fidelity micromanometer and echocardiography, respectively. Ventilation was analyzed by monitoring respiratory frequency (f(R)) and minute ventilation (V(E)) during hypoxic challenge. CIH induced a marked increase in BP, LVEDP, CO, and an increase in the slope of the ventilatory response to hypoxia. However, no difference was observed in the slopes of the ventilatory response to hypercapnia between CIH and control rats. We conclude that CIH causes significant remodeling of aortic hemodynamics. The influence of ventilatory response to hypoxia was decreased in CIH rats, suggesting a change in the neural control of breathing.Q be359ba680
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